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This article seeks to understand the public opinion formation among women of color in America. How do identity-based factors, such as linked fate with women of color (WoC), shape political evaluations? Expanding on social identity theory and intersectionality frameworks, we investigate the political influences of WoC identification and WoC intersectional linked fate. We argue that intersectional linked fate represents a deeper sense of shared experiences and interconnected outcomes across race and gender, making this both a conceptually distinct construct and a politically consequential extension of WoC identity. Further, we theorize that women of color's perceptions of intersectional linked fate drive their cross-racial support for real-world WoC in politics. We analyze data from the 2020 Collaborative Multiracial Post-Election Survey to test how intersectional linked fate correlates with evaluations of prominent WoC political figures such as Alexandria Ocasio-Cortez, Ayanna Pressley, and Michelle Obama among Black, Latina, and Asian American women. Our findings reveal that intersectional linked fate has a stronger and more consistent influence on these political evaluations than either WoC identity or racial linked fate, fostering solidarity and support for WoC politicians across racial boundaries. The results highlight the critical role of intersectional linked fate in shaping public opinion with important implications for understanding future elections, especially as candidates, politics, and the electorate continue to diversify in the United States.
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http://dx.doi.org/10.1093/poq/nfaf019 | DOI Listing |
EMBO Mol Med
September 2025
Institute for Regenerative Medicine, Medical Innovation Center and State Key Laboratory of Cardiovascular Diseases, Shanghai East Hospital, National Stem Cell Translational Resource Center & Ministry of Education Stem Cell Resource Center, Frontier Science Center for Stem Cell Research, School of Li
Primary microcephaly, a rare congenital condition characterized by reduced brain size, occurs due to impaired neurogenesis during brain development. Through whole-exome sequencing, we identified compound heterozygous loss-of-function mutations in CENTRIN 3 (CETN3) in a 5-year-old patient with primary microcephaly. As CETN3 has not been previously linked to microcephaly, we investigated its potential function in neurodevelopment in human pluripotent stem cell-derived cerebral organoids.
View Article and Find Full Text PDFMol Biomed
September 2025
National Key Laboratory of Immunity and Inflammation & Institute of Immunology, College of Basic Medical Sciences, Naval Medical University, Shanghai, 200433, China.
Dendritic cells (DCs) play a central role in coordinating immune responses by linking innate and adaptive immunity through their exceptional antigen-presenting capabilities. Recent studies reveal that metabolic reprogramming-especially pathways involving acetyl-coenzyme A (acetyl-CoA)-critically influences DC function in both physiological and pathological contexts. This review consolidates current knowledge on how environmental factors, tumor-derived signals, and intrinsic metabolic pathways collectively regulate DC development, subset differentiation, and functional adaptability.
View Article and Find Full Text PDFComp Biochem Physiol C Toxicol Pharmacol
September 2025
Department of Biological Sciences, Clemson University, Clemson, SC, USA; Clemson University Center for Human Genetics, Greenwood, SC, USA. Electronic address:
Tetrabromobisphenol A (TBBPA), a widely used flame retardant in textiles and electronics, poses toxicological risks through both environmental and indoor exposures. Biomonitoring studies have detected significant TBBPA levels in prenatal environments, including cord blood, raising concerns about developmental impacts. Using zebrafish as a model, this study addresses critical gaps in understanding how developmental TBBPA exposures perturb regulatory pathways that govern dorsoventral patterning.
View Article and Find Full Text PDFNeurosci Biobehav Rev
September 2025
Instituto de Neurobiología, Universidad Nacional Autónoma de México.
Epigenetic mechanisms are essential in neurogenesis during development and adulthood. DNA methylation, histone post-translational modifications, and non-coding RNAs regulate gene expression to maintain the neural stem cell pool and direct the fate of newborn neurons by modulating cell proliferation, migration, differentiation, maturation, and survival. Adult neurogenesis exhibits bidirectional interactions with non-social and socio-sexual factors such as sexual behavior, mate recognition, pair bonding, parental behavior, and offspring recognition.
View Article and Find Full Text PDFJ Immunother Cancer
September 2025
Department of Pediatrics, Center for Childhood Cancer and Blood Disorders, Division of Heme/Onc and Bone Marrow Transplant, Children's Hospital Colorado, University of Colorado School of Medicine, Aurora, Colorado, USA
Background: Diffuse midline glioma (DMG) and glioblastoma (GBM) are aggressive brain tumors with limited treatment options. Macrophage phagocytosis is a complex, tightly regulated process governed by competing pro-phagocytic and anti-phagocytic signals. CD47-SIRPα signaling inhibits macrophage activity, while radiotherapy (RT) can enhance tumor immunogenicity.
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