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Article Abstract
[This corrects the article DOI: 10.1039/D4RA03171G.].
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC12315996 | PMC |
| http://dx.doi.org/10.1039/d5ra90091c | DOI Listing |
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[This corrects the article DOI: 10.1039/D4RA03171G.].
View Article and Find Full Text PDFRevealing the Active Components and Therapeutic Targets of Sanao Decoction against Chronic Obstructive Pulmonary Disease Using Quantitative Analysis of Multi-Components by Single Marker and Network Pharmacology.
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As a traditional Chinese medicine, Sanao decoction (SAD) has been used to treat chronic obstructive pulmonary disease (COPD) for multi-years. However, the potential mechanism and targets for its effects of SAD remain unknown. The 94 components of SAD were identified by UPLC-LTQ-Orbitrap MS.
View Article and Find Full Text PDFHoney-fried licorice in the treatment of arrhythmia: Structure elucidation and the mechanism of antiarrhythmic activity.
Phytomedicine
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School of Pharmacy, Ningxia Medical University, Yinchuan 750004, China; Key Laboratory of Ningxia Ethnomedicine Modernization, Ministry of Education (Ningxia Medical University), Yinchuan 750004, China. Electronic address:
Aim Of The Study: To evaluate the therapeutic mechanism of Honey-fried licorice on arrhythmia, to explore the distribution of main components of Honey-fried licorice in vivo before and after processing, and to elucidate the active ingredient of Honey-fried licorice on arrhythmia.
Materials And Methods: UPLC-Q-TOF/MS were used to analyze the common and different components of raw and honey-fried licorice before and after processing. Yin deficiency syndrome was established by continuous irritability and water platform sleep deprivation, and then ventricular arrhythmia model was established by injection of calcium chloride into the tail vein.
Author Correction: One-step Conjugation of Glycyrrhetinic Acid to Cationic Polymers for High-performance Gene Delivery to Cultured Liver Cell.
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Advanced Cytometry Labs, ARC Centre of Excellence for Nanoscale BioPhotonics (CNBP), Macquarie University, Sydney, NSW, 2109, Australia.
Correction to: 18β‑Glycyrrhetinic acid altered the intestinal permeability in the human Caco‑2 monolayer cell model.
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