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Objectives: Some studies in lifespan samples show that neuroticism is associated with larger increases in negative affect (NA) with stressor exposure, whereas older age is associated with smaller differences held within older adulthood and within individuals, longitudinally. In a sample of adults over age 70, the present study (a) examined whether the relationship between stressor exposure and NA was moderated by neuroticism and (b) tested cross-sectional age differences and longitudinal aging-related change in the stressor-NA slope.
Methods: This preregistered secondary analysis draws from 3 ecological momentary assessment measurement bursts over 3 years collected in Bronx, NY. Participants (N = 318; Mage = 76.98) reported trait neuroticism at baseline, recent stressor exposure, and momentary NA 5 times daily for each 16-day burst.
Results: Using 4-level multilevel models, we found positive within- (momentary: B = 6.88, standard error [SE] = 0.44; day: B = 5.79, SE = 0.38) and between-person effects of stressor exposure (B = 19.21, SE = 3.53) on NA. Greater neuroticism was associated with higher average NA across the study (B = 4.32, SE = 1.06). Contrary to findings in younger and lifespan samples, neuroticism did not moderate the stressor-NA slope. Neither cross-sectional age differences nor longitudinal change were found in the stressor-NA slope, but longitudinal increases in average NA were observed.
Discussion: Together, these findings suggest that there are conditions when older age does not produce better emotion regulation, and that other characteristics besides neuroticism may underlie differences in stressor-related NA in older adults.
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http://dx.doi.org/10.1093/geronb/gbaf095 | DOI Listing |
Cien Saude Colet
August 2025
School of Public Health, Harvard University. Boston Estados Unidos.
In this multicenter, cross-sectional and quantitative study we evaluated the influence of urban violence and COVID-19 on the work process and team rapport of community health workers (CHWs) in eight municipalities of Northeastern Brazil. The collected information covered sociodemographics, work routines, exposure to violence, self-efficacy and coronavirus anxiety. A logistic regression was performed using as outcome variable the answer to the question: "Do you think your team work process changed during the pandemic?" The sample included 1,944 CHWs, of whom 56.
View Article and Find Full Text PDFEnviron Epidemiol
October 2025
Department of Psychiatry and Behavioral Health, The Ohio State University, Ohio.
Background: Prospective studies suggest that prenatal exposure to chemical neurotoxicants and maternal stress increase risk for psychiatric problems. However, most studies have focused on childhood outcomes, leaving adolescence-a critical period for the emergence or worsening of psychiatric symptoms-relatively understudied. The complexity of prenatal coexposures and adolescent psychiatric comorbidities, particularly among structurally marginalized populations with high exposure burdens, remains poorly understood.
View Article and Find Full Text PDFFront Psychiatry
August 2025
Department of Psychiatry, Nagoya City University East Medical Center, Nagoya, Japan.
Although post-earthquake psychological distress arises from a complex interplay of personal vulnerabilities and environmental stressors, the pathways by which these factors interact remain underexplored. We surveyed 327 hospital nurses in Nanao City, Japan, approximately eight months after the magnitude-7.5 2024 Noto Peninsula earthquake; 224 complete responses were analyzed.
View Article and Find Full Text PDFAlpha Psychiatry
August 2025
Department of Pharmacology, School of Pharmaceutical Science, Ohu University, Koriyama, 963-8611 Fukushima, Japan.
Objective: Hypothalamic‒pituitary‒adrenal axis response is essential for coping with acute stressors, while maladaptive stress coping may increase the risk of major depressive disorder. We previously demonstrated that behavioral patterns induced by prior psychological stress predict coping levels in response to future stressors. This study investigated whether activating corticotropin-releasing hormone (CRH) and corticosteroid receptors mediates psychological stress-induced coping behavior.
View Article and Find Full Text PDFProc Natl Acad Sci U S A
September 2025
Life Science Center for Survival Dynamics, Tsukuba Advanced Research Alliance, University of Tsukuba, Tsukuba 305-8577, Japan.
All organisms are exposed to various stressors, which can sometimes lead to organismal death, depending on their intensity. While stress-induced organismal death has been observed in many species, the underlying mechanisms remain unclear. In this study, we investigated the molecular mechanisms of stress-induced organismal death in the fruit fly .
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