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Article Abstract
A recent in vitro study showed that pharmacological inhibition of the nuclear export receptor XPO1 suppresses oxaliplatin-induced nuclear release of HMGB1 and HMGB2, as well as the translocation of CALR to the plasma membrane. Moreover, cell-targeted-HMGB2 protein potently induced CALR exposure, even in the absence of oxaliplatin.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC11520571 | PMC |
| http://dx.doi.org/10.1080/2162402X.2024.2421028 | DOI Listing |
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