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Calcification following tongue necrosis induced by vasopressor use in a nonintubated patient with septic shock: a case report. | LitMetric

Calcification following tongue necrosis induced by vasopressor use in a nonintubated patient with septic shock: a case report.

J Med Case Rep

Division of Pulmonology and Critical Care Medicine, Department of Internal Medicine, College of Medicine, Chungnam National University, 282 Munhwa-Ro, Jung-Gu, Daejeon, 35015, Republic of Korea.

Published: September 2024


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Article Abstract

Background: Tongue necrosis is a rare and relatively uncommon condition, usually caused by vasculitis, thrombosis, severe hypotension due to septic or cardiogenic shock, vasopressor use, or intubation. Following damage such as necrosis, dystrophic calcification, a type of soft tissue calcification, can occur.

Case Presentation: Herein, we present a unique case of bilateral tongue necrosis in a patient with nonintubated septic shock. A 70-year-old East Asian man with no significant medical history presented to the emergency department with postprandial epigastric pain. The patient was admitted to the intensive care unit with hypotension due to septic shock and disseminated intravascular coagulation. After a short course of vasopressors, the patient developed tongue discoloration and swelling without limb ischemia. Computed tomography was performed to observe the tongue necrosis, and calcification of the tongue was found. The patient was successfully treated by wiping the area with a hexamidine-soaked gauze.

Conclusion: Tongue necrosis remains a rare finding, and its occurrence as a complication of vasopressor use is even rarer. Therefore, even with relatively short courses of vasopressors in the intensive care unit, daily visualization of the tongue to check for discoloration, along with daily inspection and pulse checks of the limbs, can help identify vasospasms. These measures allow for prompt intervention, minimizing permanent damage and shortening the recovery time.

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Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC11412059PMC
http://dx.doi.org/10.1186/s13256-024-04796-6DOI Listing

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