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Two strains of Japanese quail (Coturnix coturnix japonica), including 250 birds of desert and 250 birds of white color, all one day old and unsexed, were divided into five treatment groups with 50 replicates in each group. These treatments included five levels of metabolism energy (ME) levels, including 2700, 2800, 2900, 3000, and 3100 Kcal/Kg diet. The study included one stage from day 1 to day 42 of birds' age. The results confirmed a statistically significant difference (≤0.05) caused by ME levels in the body weight, weight gain, feed conversion ratio (feed [g] weight gain [g]), water consumption, water conversion ratio (ml weight gain [g]), protein conversion ratio (protein [g] weight gain [g]), energy conversion ratio (Kcal weight gain [g]), carcass weight, in addition to albumin and triglyceride. Therefore, the results showed significant effects (≤0.05) of ME levels and the interaction on feed consumption, protein consumption, edible giblet percentage, tenderness, and juiciness. Significant differences (≤0.05) were also caused by ME levels in the total cholesterol. In addition, significant differences (≤0.05) have been found in the interaction on mortality percentage. Net return (Iraqi Dinar/live weight [Kg]) for desert quail was better than that for the white quail (2900 Kcal/Kg diet), and the interaction effect was stronger on the desert strain with 2900 Kcal than the white strain.
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http://dx.doi.org/10.22092/ARI.2022.359474.2428 | DOI Listing |
Methods Cell Biol
September 2025
Department of Basic Sciences, Faculty of Medicine and Sciences, Universidad San Sebastián, Santiago, Chile. Electronic address:
Obesity is a multifactorial disease characterized by excessive accumulation of adipose tissue, resulting from an imbalance between energy intake and expenditure. Mouse models have emerged as invaluable tools for elucidating the complex genetic, environmental, and physiological mechanisms driving to obesity. This chapter provides an overview of the methodologies employed to establish and study obesity in mice, highlighting their relevance to human disease.
View Article and Find Full Text PDFMethods Cell Biol
September 2025
Centre de Recherche des Cordeliers, Equipe Labellisée par la Ligue Contre le Cancer, Université de Paris Cité, Sorbonne Université, Inserm U1138, Institut Universitaire de France, Paris, France; Metabolomics and Cell Biology Platforms, UMS AMICCa, Gustave Roussy, Villejuif, France. Electronic ad
Cushing's syndrome is caused by chronic exposure to excessive levels of glucocorticoids. It is characterized by significant phenotypic alterations including increased visceral adiposity and fat deposits on the cheeks, leading to a characteristic 'moon face' appearance. Although glucocorticoid therapy is widespread, its associated side effects are of significant clinical concern.
View Article and Find Full Text PDFFitoterapia
September 2025
Yunnan Key Laboratory of Southern Medicine Utilization, College of Traditional Chinese Medicine, Yunnan University of Chinese Medicine, Kunming 650500, China. Electronic address:
The gut microbiota and its products are recognized as pivotal contributors to the pathogenesis of metabolic-associated fatty liver disease (MAFLD). Shenling Jianpiwei formula (SLJPW), a prescription renowned for its protective effects in intestinal disorders, demonstrates efficacy against MAFLD. However, its underlying mechanisms and chemical composition remain unclear.
View Article and Find Full Text PDFDiabetes Metab Syndr
September 2025
School of Nursing, Capital Medical University, Beijing, China. Electronic address:
Introduction: Gestational diabetes mellitus (GDM) is a common pregnancy complication, with a significant risk of recurrence in subsequent pregnancies.
Aims: We aimed to explore the incidence and risk factors associated with the recurrence of GDM among women with a history of GDM.
Methods: We searched several databases (PubMed, Embase, Web of Science, Cochrane Library, Ovid, CINAHL, ProQuest, China Knowledge Resource Integrated Database, Wanfang Database, VIP Database, and China Biology Medicine Database) from January 1961 to June 2024.
PLoS One
September 2025
Department of Anatomy and Physiology, The University of Melbourne, Parkville, Victoria, Australia.
Food intake is a key regulator of the digestive system function; however, little is known about organ- and sex-specific differences in food-driven regulation. We placed male and female C57Bl/6 mice on time-restricted feeding (TRF), limiting access to food to an 8-hour window. Food was added either at dark (ZT12) or light (ZT0) onset for 14 days.
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