Paraquat Poisoning.

J Assoc Physicians India

Director, Department of Internal Medicine, Fortis Escorts Hospital, Jaipur, Rajasthan, India; Corresponding Author.

Published: April 2023


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To conduct a visual analysis of the papers published in the Chinese Journal of Industrial Hygiene and Occupational Diseases since its inception, and to understand the current situation and development trends in the field of occupational health and occupational diseases. A total of 9894 papers published in the Chinese Journal of Industrial Hygiene and Occupational Diseases from January 1, 1983 to December 31, 2023 were retrieved from China National Knowledge Infrastructure (CNKI) in February 2024. Non-academic literature was screened and excluded.

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Paraquat, a commonly available herbicide, when consumed in high doses, affects organs with high blood flow (lungs, heart, kidney and liver), leading to pulmonary fibrosis, respiratory failure and death. Few reports of rescue lung transplantation exist. Complete depletion of paraquat from the body is necessary prior to transplant; however, timing for and concerns after lung transplantation remain unknown.

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Introduction: Paraquat poisoning remains of toxicological concern as it may cause multiple organ failure after substantial ingestion, and in those less severely poisoned, pulmonary fibrosis.

Case Summary: We describe a patient who developed paraquat-induced pulmonary fibrosis and iatrogenic atelectasis due to a right subclavian vein catheterization.

Images: The chest radiograph obtained on hospital day 3 demonstrates a right tension hemothorax.

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Beneficial effects of dihydrocapsaicin on paraquat-induced acute kidney injury: role of Sox9/Sesn2 axis-mediated autophagy.

Ecotoxicol Environ Saf

August 2025

Department of Emergency Medicine, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China. Electronic address:

Autophagy inhibition induced by paraquat (PQ) poisoning is an important factor causing multi-organ damage, including renal tissue. Dihydrocapsaicin (DHC), the main active ingredient in chili peppers, was reported to induce autophagy, but its effect on PQ-induced AKI remains unclear. Rats were intraperitoneally injected with 25 mg/kg PQ to establish AKI rat model followed immediately by DHC administration (2.

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