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Article Abstract
[This corrects the article DOI: 10.1016/j.ajpc.2021.100151.].
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC8984522 | PMC |
| http://dx.doi.org/10.1016/j.ajpc.2021.100200 | DOI Listing |
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Corrigendum to Synopsis of an integrated guidance for enhancing the care of familial hypercholesterolaemia: An Australian perspective [American Journal of Preventive Cardiology 6 (2021) 100151].
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Article Synopsis
- The text mentions a correction to a previously published article with the DOI: 10.1016/j.ajpc.2021.100151.
- This correction suggests that there were errors or inaccuracies in the original article that needed to be addressed.
- The updated information is important for researchers and readers who rely on the findings of that specific study.
Corrigendum to "Role of UHRF1 in malignancy and its function as a therapeutic target for molecular docking towards the SRA domain" [Int. J. Biochem. Cell Biol. 114 (September 2019) 105558].
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Cancer pathogenesis has been attributed to the minor and major disruptions in the cell cycle, with a key role being played by several of the recently discovered epigenetic factors. Lately, UHRF1 (Ubiquitin-like with containing PHD and RING Finger domains 1), an epigenetic regulator has been shown to be evidently over expressed in numerous malignancies through an in-depth review of literature. Molecular docking studies have found that existing drugs such as propranolol, naphthazarin and thymoquinone have favourable interactions with specific domains of UHRF1.
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