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Objective: To investigate biomechanics, muscle performance, and disability of the craniocervical region in temporomandibular disorder (TMD) patients and compare them with controls.
Methods: Craniocervical posture was evaluated using lateral photography and radiography. Range of motion, muscle performance, disability, and TMD severity were assessed using an inclinometer, Functional Strength Testing of Cervical Spine, Neck Disability Index, and Fonseca Anamnestic Index, respectively.
Results: Compared to the control group, the TMD group demonstrated higher cervical flexion angle (=0.005) and neck disability (<0.001) as well as lower cervical extension (=0.040), right cervical rotation (=0.005), left cervical rotation (<0.001), and tragus-C7-horizontal (=0.048) angles, and reduced muscle performances (≤0.001). Most patients had higher than normal craniocervical angle (<0.001). Muscle performance in each cervical motion (<0.005) and disability (<0.001) were associated with TMD severity in the TMD group.
Conclusion: Biomechanics, muscle performance, and disability of craniocervical region were altered in the TMD group.
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http://dx.doi.org/10.1080/08869634.2021.1938854 | DOI Listing |
Eur J Sport Sci
October 2025
University Jean Monnet Saint-Etienne, Lyon 1, University Savoie Mont-Blanc, Inter-university Laboratory of Human Movement Biology, EA 7424, F-42023, Saint-Etienne, France.
The purpose of this study was to evaluate the evolution of jump and sprint force-production capacities with maturation in young soccer players. One hundred sixteen young elite male soccer players aged 11-17 years were assigned to six different groups according to their maturity status. The force-velocity (F-V) profiles in jumping and sprinting performances were compared among groups.
View Article and Find Full Text PDFMed Sci Monit
September 2025
Department of Anesthesiology and Intensive Care, Faculty of Medicine, Collegium Medicum University of Warmia and Mazury, Olsztyn, Poland.
Modern anesthesia, intensive care, and emergency medicine rely heavily on neuromuscular blocking agents (NMBAs), first introduced in 1942. These agents not only facilitate endotracheal intubation but also improve surgical conditions by suppressing muscle responses to stimuli. NMBAs function via depolarizing (eg, succinylcholine) or non-depolarizing mechanisms.
View Article and Find Full Text PDFMagn Reson Med
September 2025
Department of Medical Physics, Memorial Sloan Kettering Cancer Center, New York, New York, USA.
Purpose: To develop and validate a fast, motion-robust, free-breathing abdominal 3D T1ρ mapping method by combining variable-density diamond radial k-space sampling with fast-MAPSS (magnetization-prepared angle-modulated partitioned-k-space spoiled gradient-echo snapshots).
Methods: 3D MAPSS T1ρ imaging was performed at 3T using five spin-lock time (TSL) pairs in phantom scans and three TSL pairs in nine healthy volunteers. Phantom experiments compared Cartesian sampling (reference) with stack-of-stars and diamond radial sampling.
J Cachexia Sarcopenia Muscle
September 2025
Integrative Muscle Biology Laboratory, Division of Rehabilitation Sciences, College of Health Professions, University of Tennessee Health Science Center, Memphis, Tennessee, USA.
Background: Cancer promotes muscle wasting through an imbalance in the tightly regulated protein synthesis and degradation processes. An array of intracellular signalling pathways, including mTORC1 and AMPK, regulate protein synthesis, and these pathways are responsive to the muscle's microenvironment and systemic stimuli. Although feeding and fasting are established systemic regulators of muscle mTORC1 and protein synthesis, the cancer environment's impact on these responses during cachexia development is poorly understood.
View Article and Find Full Text PDFCell Commun Signal
September 2025
Department of Cytology, Institute of Anatomy, Medical Faculty, Ruhr-University Bochum, Universitätsstr. 150, Building MA 5/52, Bochum, 44801, Germany.
Background: Amyotrophic lateral sclerosis (ALS) is a devastating neurodegenerative disease characterized by oxidative stress and progressive motor neuron degeneration. This study evaluates the potential neuroprotective effects of caffeine in the Wobbler mouse, an established model of ALS.
Methods: Wobbler mice received caffeine supplementation (60 mg/kg/day) via drinking water, and key parameters, including muscle strength, NAD metabolism, oxidative stress, and motor neuron morphology, were assessed at critical disease stages.