Category Ranking
98%
Total Visits
921
Avg Visit Duration
2 minutes
Citations
20
Article Abstract
To understand the microbial social behaviors regulated by acyl-homoserine lactones (AHLs) in the upflow anaerobic sludge blanket (UASB) during the restored process after a shock loading, the correlation analyses of AHLs and components of extracellular polymeric substances (EPS), AHLs genes and microbes, and AHLs and microbes were investigated. The results showed that the performance could be restored by regulating influent organic loading rate stage-by-stage. A variation in microbial community and endogenous AHLs was also found during the restoration process. It was found that C-HSL had improved the synthesis of protein in EPS and resulted in better aggregation of microbes. C-HSL, as well as C-HSL and 3-oxoC-HSL, could prompt the metabolism of acidogenic fermentation bacteria. While 3-oxoC-HSL was identified as the key signal molecule in enhancing methanogenesis. The present work advanced the understanding of microbial social behaviors and provided an attractive strategy for the restoration of anaerobic digestion after shock loadings.
Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1016/j.biortech.2021.124972 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Exploration of the social determinants of diarrhoea, rotavirus vaccine uptake, and vaccine 'fatigue' in Ethiopia, Kenya, and Malawi.
PLoS One
September 2025
Department of Social Science, Malawi Liverpool Wellcome Clinical Programme, Blantyre, Malawi.
Diarrhoea due to rotavirus remains a significant cause of child mortality in developing regions. Caregivers' perspectives on the social determinants of gastroenteritis and childhood vaccination, including the rotavirus vaccine, were explored through focus group discussions in Ethiopia (n = 6), Kenya (n = 14), and Malawi (n = 10), using a combination of thematic and framework analysis approaches. The results show that diarrhoea was perceived to be a burden in all three countries, particularly among infants, due to challenges in WASH (water, sanitation, and hygiene) infrastructures and poverty.
View Article and Find Full Text PDFPublic views on xenotransplantation from the first representative sample survey in the UK.
Lancet
September 2025
Policy Innovation and Evaluation Research Unit, Department of Health Services Research and Policy, London School of Hygiene & Tropical Medicine, London WC1H 9SH, UK.
The gut commensal attenuates indole-AhR signaling and restores ASD-like behaviors with BTBR mice.
Front Microbiol
August 2025
State Key Laboratory of Microbial Diversity and Innovative Utilization, Institute of Microbiology, Chinese Academy of Sciences, Beijing, China.
Autism spectrum disorders (ASD), a group of neurodevelopmental disorders characterized by the core symptoms of impaired social communication and stereotyped behaviors, is strongly associated with dysregulated microbiota-gut-brain axis. Emerging evidence suggests that , which showed reduced abundance in ASD cohorts, holds therapeutic potential, though its interaction with host remain unexplored. Here, we investigated the efficacy and molecular basis of 4P-15 (4P-15) in BTBR /J (BTBR) mice, an idiopathic ASD mouse model.
View Article and Find Full Text PDFA Multimodal Observational Case-Control Study Exploring Gut Microbiota-Hippocampus Alterations in Individuals With High Positive Schizotypy From the General Population.
Biol Psychiatry Glob Open Sci
November 2025
University of Basel, Department of Clinical Research (DKF), University Psychiatric Clinics, Translational Neurosciences, Basel, Switzerland.
Background: The hippocampus plays a critical role in psychosis, with reduced volume observed across the psychosis continuum. These structural changes are associated with cognitive deficits, symptom severity, and increased risk of psychosis progression. Elevated hippocampal perfusion and glutamate/GABA (gamma-aminobutyric acid) imbalance further suggest metabolic dysregulation as a key mechanism.
View Article and Find Full Text PDFObservational analysis of biological remission as a treatment target for severe asthma: UK severe asthma registry.
J Allergy Clin Immunol Pract
September 2025
Wellcome Wolfson Institute for Experimental Medicine, Queen's University Belfast, UK; Belfast Health and Social Care Trust, Belfast, UK.
Background: The aim of biologic therapies in severe asthma is inhibition of T2 inflammatory pathways.
Objective: We hypothesized that patients who achieve complete suppression of IL-5 & IL4/IL13 pathways with biologic therapy (FeNO <20ppb & blood eosinophil count (BEC) <0.15x10ˆ9, 'biological remission') would have better outcomes than patients with incomplete suppression of T2 biology.