Category Ranking
98%
Total Visits
921
Avg Visit Duration
2 minutes
Citations
20
Article Abstract
Download full-text PDF |
Source |
|---|---|
| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6495314 | PMC |
| http://dx.doi.org/10.1016/j.mjafi.2018.03.012 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Detour of bile acid routes as therapeutic roadmap for cholemic nephropathy.
J Hepatol
February 2024
Department of Medicine, Division of Gastroenterology and Hepatology, Medical University Graz, Austria. Electronic address:
Bile Cast Nephropathy Due to Hepatitis A-induced Hyperbilirubinemia: A Case Report and Literature Review.
Cureus
March 2023
Internal Medicine, The Wright Center for Graduate Medical Education, Scranton, USA.
Bile cast nephropathy (BCN) or cholemic nephrosis (CN) is a form of acute renal dysfunction that occurs in the setting of hepatic dysfunction and hyperbilirubinemia. We present a case of a 58-year-old woman with a four-day history of intractable nausea, vomiting, and yellowish discoloration of her skin and eyes. Laboratory workup was notable for elevated total bilirubin (mainly direct), liver enzymes, creatinine, and blood urea nitrogen (BUN).
View Article and Find Full Text PDFCholemic Nephrosis: An Autopsy Study of a Forgotten Entity.
Turk Patoloji Derg
December 2021
Department of Pathology, Indira Gandhi Medical College and Research Institute, PUDUCHERRY, INDIA.
Objective: The aim of the study is to do a clinicopathologic study of post mortem kidney biopsies with significant deposition of bilirubin pigment within tubular epithelial cells and in the lumen of distal tubules as a bile cast.
Material And Method: All post mortem specimens with acute tubular necrosis, with the presence of bile casts in tubules or bile pigment deposition in the tubular epithelium during the period 2015-2018 were examined for gross and histopathology along with biochemical parameters and viral markers.
Results: Bile casts with sloughed renal tubular epithelial cells and occasional macrophages were present in the distal convoluted tubule in 78.
Pathological Study of Facial Eczema (Pithomycotoxicosis) in Sheep.
Animals (Basel)
April 2021
Departamento de Sanidad Animal, Facultad de Veterinaria, Universidad de León, C/Prof. Pedro Cármenes s/n, E-24071 León, Spain.
Facial eczema (FE) is a secondary photosensitization disease of farm ruminants caused by the sporidesmin A, produced in the spores of the saprophytic fungus . This study communicates an outbreak of ovine FE in Asturias (Spain) and characterizes the serum biochemical pattern and the immune response that may contribute to liver damage, favoring cholestasis and the progression to fibrosis and cirrhosis. Animals showed clinical signs of photosensitivity, with decrease of daily weight gain and loss of wool and crusting for at least 6 months after the FE outbreak.
View Article and Find Full Text PDFHepatorenal syndrome: a historical appraisal of its origins and conceptual evolution.
Kidney Int
June 2021
Division of Nephrology and Hypertension, University of Miami Miller School of Medicine, Miami, Florida, USA.
The hepatorenal syndrome (HRS), a progressive but potentially reversible deterioration of kidney function, constitutes a serious complication of hepatic decompensation. Coexistence of liver/kidney damage, mentioned in the dropsy literature, was highlighted by Richard Bright in 1827 and confirmed in 1840 by his contemporary nephrology pioneer Pierre Rayer. Cholemic nephrosis was described in 1861 by Friedrich Frerichs, and the renal tubular lesions of HRS by Austin Flint in 1863.
View Article and Find Full Text PDF