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Background: Accurate varicella (chickenpox) and zoster (shingles)-coded hospitalisation data are important for disease surveillance and to examine the impact of immunisation programs. Varicella and zoster generally occur many years apart, with hospital episodes coded for both diseases concurrently implausible.
Objective: We aimed to describe varicella-zoster virus-related hospital episodes coded for both varicella and zoster or postherpetic neuralgia ('dual-coded') in Australia.
Post-herpetic abdominal pseudohernia (PHAP) is a rare complication associated with herpes zoster (HZ), characterized by transient paresis of the myotome ipsilateral to the affected segmental nerves. Clinically, it manifests as asymptomatic laxity and protrusion of the abdominal musculature, mimicking abdominal wall herniation. While the varicella-zoster virus (VZV) primarily affects the sensory nervous system, the incidence and prognosis of motor involvement remain poorly estimated.
View Article and Find Full Text PDFHerpes zoster peripheral nerve complications: Their pathophysiology in spinal ganglia and nerve roots.
Intractable Rare Dis Res
November 2023
Department of Neurology, Aomori Prefectural Central Hospital, Aomori, Japan.
Varicella zoster virus (VZV) causes chickenpox at the primary infection and then becomes latent in the spinal dorsal root ganglia; VZV can reactivate with aging, immunosuppression, stress, and other factors, occurring as herpes zoster (HZ) at 1-2 skin segments. HZ peripheral nerve complications caused by VZV reactivation include Hunt syndrome, segmental HZ paresis, post-herpetic neuralgia, and Guillain-Barré syndrome (GBS). We have encountered the rare HZ complications of upper-limb paresis, myeloradiculitis, and polyradiculoneuritis: an adult woman with upper-limb paresis consistent with the nerve root on segments above the thoracic HZ dermatome; another woman exhibiting ascending myeloradiculitis originating at the Th11-12 roots; an elderly woman with ascending VZV polyradiculoneuritis resembling GBS; an adult with VZV quadriplegia with disseminated HZ; and an elderly patient with VZV-associated polyradiculoneuritis.
View Article and Find Full Text PDFBackground Herpes zoster is a common viral infection caused by reactivation of the varicella-zoster virus (VZV) characterized by the presence of a segmental distribution of painful grouped vesicles on an erythematous base. It is associated with several complications like zoster-associated pain (ZAP), postherpetic neuralgia (PHN), pigmentary changes, scarring, secondary infections, and dermatosis as well as severe systemic complications. Aims/objectives The aim of the study was to analyze the various clinical and epidemiological patterns of herpes zoster and post-herpetic complications.
View Article and Find Full Text PDFThe Spectrum of Neurological Manifestations of Varicella-Zoster Virus Reactivation.
Viruses
July 2023
School of Psychology and Neuroscience, College of Medical, Veterinary & Life Sciences, Garscube Campus, University of Glasgow, Glasgow G61 1QH, Scotland, UK.
Varicella-Zoster virus (VZV) is a pathogenic human alpha herpes virus that causes varicella (chicken pox) as a primary infection and, following a variable period of latency in different ganglionic neurons, it reactivates to produce herpes zoster (shingles). The focus of this review is on the wide spectrum of the possible neurological manifestations of VZV reactivation. While the most frequent reactivation syndrome is herpes zoster, this may be followed by the serious and painful post-herpetic neuralgia (PHN) and by many other neurological conditions.
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