[Retracted] lncRNA SNHG3 promotes breast cancer progression by acting as a miR‑326 sponge.

Following the publication of the above article, the authors have requested that it be retracted. Subsequently to having performed these experiments, the authors have realized that their MCF‑7 cells had become contaminated while performing the colony formation and transwell experiments. Furthermore, they were unable to reproduce the results from the experiments, and knockdown of was found not to affect tumor growth in nude mice.

lncRNA SNHG3 promotes breast cancer progression by acting as a miR‑326 sponge.

Accumulating evidence suggests that long noncoding RNA (lncRNA) small nucleolar RNA host gene 3 (SHNG3) plays crucial roles in the initiation and progression of various types of malignant cancers. Yet, the role played by SNHG3 in breast cancer as well as the associated mechanisms remain largely unclear. The expression of SNHG3 was detected in breast cancer tissues and cell lines by reverse‑transcription quantitative PCR (RT‑qPCR).

Characterization and imaging of surgical specimens of invasive breast cancer and normal breast tissues with the application of Raman spectral mapping: A feasibility study and comparison with randomized single-point detection method.

A mapping technique was used in the present study to explore the biological and imaging characteristics of invasive breast cancer and normal breast tissues in Raman examination data and construct a diagnostic model for breast cancer. Raman examination data reflect the biochemical or molecular characteristics of the target tissues. A total of 45 specimens from patients with breast cancer who underwent surgery and 25 adjacent normal breast tissue specimens were included in the present study.

Functional analyses of microRNA-326 in breast cancer development.

MicroRNA-326 (miR-326) was reported to be dysregulated and involved in the progression of multiple cancers. However, the clinical significance, biological role and underlying mechanism of miR-326 in the carcinogenesis of breast cancer are still unclear. In the present study, we showed that miR-326 was down-regulated in human breast cancer tissues and cell lines.