Negatively Regulates Virulence via Enhancing Effector Expression and Suppressing Host Immune Responses.

The soil-borne fungal pathogen causes devastating vascular wilt disease in numerous crops, including cotton. In this study, we reveal that , a highly conserved sarcosine oxidase gene, is significantly upregulated during host infection and plays a multifaceted role in fungal physiology and pathogenicity. Functional deletion of leads to increased fungal virulence, accompanied by enhanced microsclerotia formation, elevated carbon source utilization, and pronounced upregulation of effector genes, including over 50 predicted secreted proteins genes.

Integrative identification of key genes governing wilt resistance in using machine learning and WGCNA.

Introduction: wilt, caused by , is one of the most devastating diseases affecting global cotton () production. Given the limited effectiveness of chemical control measures and the polygenic nature of resistance, elucidating the key genetic determinants is imperative for the development of resistant cultivars. In this study, we aimed to dissect the temporal transcriptional dynamics and regulatory mechanisms underlying response to infection.

Emissions of air pollutants from non-road construction machinery in Beijing from 2015 to 2019.

Construction machinery (CM) is considered to be a significant source of air pollution. The estimation of emissions from CM is essential for policy-makers to control air pollution and reduce carbon emissions. In this study, air pollutant emissions from CM with four emissions standards (Beijing I-IV) in Beijing from 2015 to 2019 were estimated by combining data from the Non-road Mobile Source Emissions Inventory Compiled Technical Guidelines, Motor Vehicle Emission Simulator (MOVES) and actual measurements.