Ascorbate and glutathione independently alleviate arsenate toxicity in brinjal but both require endogenous nitric oxide.

In this study, we have explored the possible role of ascorbic acid (ASC) and glutathione (GSH) in alleviating arsenate (As ) toxicity in brinjal roots. Moreover, we have also focused our attention on the possible involvement of endogenous nitric oxide (NO) in accomplishing this task. As treatment negatively impacts the length and fresh weight of roots and shoots as well as the dry weight and fitness of roots, and this was accompanied by greater As accumulation in roots and shoots of brinjal.

Regulation of ascorbate-glutathione cycle by exogenous nitric oxide and hydrogen peroxide in soybean roots under arsenate stress.

The role of nitric oxide (NO) and hydrogen peroxide (HO) is well known for regulating plant abiotic stress responses. However, underlying mechanisms are still poorly understood. Therefore, the present study investigated the involvement of NO and HO signalling in the regulation of arsenate toxicity (As) in soybean roots employing a pharmacological approach.

Mitigation of arsenate toxicity by indole-3-acetic acid in brinjal roots: Plausible association with endogenous hydrogen peroxide.

The role of indole-3-acetic acid (IAA) and hydrogen peroxide (HO) crosstalk in regulating metal stress is still less known. Herein, role of IAA in alleviating arsenate (As) toxicity in brinjal seedlings along with its probable relation with endogenous HO was investigated. Arsenate hampered root growth due to greater accumulation of As and decrease in phosphorus uptake that resulted into inhibited photosynthesis and cell death.

Mitigation of chromium (VI) toxicity by additional sulfur in some vegetable crops involves glutathione and hydrogen sulfide.

Toxic metals cause substantial reduction in crop yields every year. Therefore, worldwide scientific efforts are being made to reduce such losses in crop productivity by using certain chemical protectants such as nutrients like sulfur (S), hydrogen sulfide (HS), glutathione (GSH), etc. Therefore in this study, we have tested potential of additional S, along with probable involvement of HS and GSH in mitigating hexavalent chromium (CrVI) toxicity in tomato, pea and brinjal seedlings.

Silicon tackles butachlor toxicity in rice seedlings by regulating anatomical characteristics, ascorbate-glutathione cycle, proline metabolism and levels of nutrients.

Reckless use of herbicides like butachlor (Buta) in the fields represents a serious threat to crop plants, and hence to their productivity. Silicon (Si) is well known for its implication in the alleviation of the effects of abiotic stresses; however, its role in mitigating Buta toxicity is not yet known. Therefore, this study was carried out to explore the role of Si (10 µM) in regulating Buta (4 µM) toxicity in rice seedlings.

Full sunlight acclimation mechanisms in Riccia discolor thalli: Assessment at morphological, anatomical, and biochemical levels.

Light occupies a central position in regulating development of plants. Either little or excess of light could be harmful for plants. Since bryophytes are shade loving organisms, they must adapt to function in fluctuating light regimes.

Ascorbic acid is essential for inducing chromium (VI) toxicity tolerance in tomato roots.

Problem of chromium (Cr) pollution is of great scientific concern as it adversely affects crop productivity worldwide. Therefore, scientific efforts are being made to minimize Cr toxicity in crop plants by using various methods. Of these methods, use of certain chemicals like ascorbic acid (ASC), glutathione, proline, nutrients, etc.

Dose dependent differential effects of toxic metal cadmium in tomato roots: Role of endogenous hydrogen sulfide.

In this study, hydroponic experiments were conducted to elucidate mechanism(s) that are associated with differential effects of low (5 μM) and high (25 μM) dose of cadmium (Cd) stress in tomato. Furthermore, emphasis has also been focused on any involvement of endogenous hydrogen sulfide (HS) in differential behaviour of low and high doses of Cd stress. At low dose of Cd, root growth i.

Nitric oxide-mediated regulation of sub-cellular chromium distribution, ascorbate-glutathione cycle and glutathione biosynthesis in tomato roots under chromium (VI) toxicity.

Unprecedented anthropogenic activities have led to contamination of soil and water with toxic metals that present a great threat to crop yields. This situation has compelled researchers to understand metal toxicity responses in order to develop strategies to curtail toxic metal-mediated losses in crop yields. Past decade has witnessed tremendous developments with regard to the role of nitric oxide (NO) in regulating abiotic stresses including toxic metal in crop plants.

New adventitious root formation and primary root biomass accumulation are regulated by nitric oxide and reactive oxygen species in rice seedlings under arsenate stress.

Nitric oxide (NO) and reactive oxygen species (ROS) are important signaling molecules regulating development of plants. However under metal stress, in developmental processes of plants their implications are not largely known. Therefore, in the present study, role of NO and ROS crosstalk in the regulation of formation of new adventitious roots (NARs) and primary root biomass accumulation (PRBA) has been investigated in rice seedlings under arsenate (As) stress.

Sulphur alters chromium (VI) toxicity in Solanum melongena seedlings: Role of sulphur assimilation and sulphur-containing antioxidants.

The present study investigates modulation in hexavalent chromium [Cr(VI) 25 μM] toxicity by sulphur (S; 0.5, 1.0 and 1.