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Cyclone disasters pose significant health challenges, including severe hematological emergencies resulting from trauma, infections, and systemic inflammatory responses. Injuries sustained during cyclones can lead to trauma-induced coagulopathy, acute anemia, and disseminated intravascular coagulation (DIC), requiring immediate intervention. Additionally, post-disaster conditions such as malnutrition, dehydration, and poor sanitation contribute to increased susceptibility to infections, further exacerbating blood-related disorders. Hematologists play a vital role in diagnosing and managing these complications through rapid hematological assessments, transfusion support, and coagulation monitoring. Disaster preparedness is crucial in mitigating the impact of cyclone-induced hematological disorders. Ensuring an adequate blood supply, establishing emergency transfusion protocols, and collaborating with disaster response teams are essential pre-disaster strategies. Post-cyclone responses require hematologists to triage affected individuals, manage thromboembolic risks, and monitor sepsis-related hematological dysfunctions. Long-term rehabilitation efforts should focus on nutritional support, anemia management, and continuous surveillance of blood disorders in displaced populations to prevent chronic hematological complications.
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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC12401405 | PMC |
http://dx.doi.org/10.1097/MS9.0000000000003475 | DOI Listing |
Ann Med Surg (Lond)
September 2025
Department of Biomedical and Laboratory Science, Africa University, MUutare, Zimbabwe.
Cyclone disasters pose significant health challenges, including severe hematological emergencies resulting from trauma, infections, and systemic inflammatory responses. Injuries sustained during cyclones can lead to trauma-induced coagulopathy, acute anemia, and disseminated intravascular coagulation (DIC), requiring immediate intervention. Additionally, post-disaster conditions such as malnutrition, dehydration, and poor sanitation contribute to increased susceptibility to infections, further exacerbating blood-related disorders.
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