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Weekend discharge delays remain a significant contributor to prolonged hospital length of stay, often resulting from reduced availability of consultants, imaging, case management, and therapy services on Saturdays and Sundays. Expanding hospital operations to full seven-day coverage is often proposed. However, such solutions are frequently cost-prohibitive and have shown inconsistent benefits in general inpatient settings. This review proposes a more practical and scalable alternative that focuses on leveraging Thursday and Friday as high-impact planning days to mitigate weekend-related discharge bottlenecks. Through a narrative review of 15 studies published between 2010 and 2024, we identified key operational strategies, including early multidisciplinary rounds, Friday discharge huddles, and coordination guided by standardized discharge checklists, that enable teams to proactively clear discharge barriers before the weekend. These interventions improve weekend discharge volume, reduce unnecessary hospital days, and enhance patient flow without requiring full weekend staffing models. In addition, embedding case management within the emergency department can reduce avoidable weekend admissions by directing low-acuity patients toward outpatient services and follow-up. Together, these targeted weekday efforts form a cost-effective, immediately actionable framework that hospitals can adopt to reduce weekend delays, improve efficiency, and lower overall length of stay without compromising safety or quality of care.
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http://dx.doi.org/10.7759/cureus.87526 | DOI Listing |
Eur J Case Rep Intern Med
August 2025
Department of Internal Medicine, Dubai Hospital, Dubai Health, Dubai, Dubai, United Arab Emirates.
Introduction: Primary central nervous system vasculitis (primary CNS vasculitis) is a rare inflammatory disorder that affects small-to-medium-sized cerebral vessels, often leading to recurrent strokes. Diagnosis is vague due to non-specific neurological symptoms. Imaging findings, cerebrospinal fluid (CSF) analysis and exclusion of systemic vasculitis are essential for diagnosis.
View Article and Find Full Text PDFCureus
September 2025
Internal Medicine, NYU Langone Hospital - Long Island, Mineola, USA.
Drug-induced immune thrombocytopenia (DITP) is a rare but life-threatening condition characterized by a sudden and serious drop in the number of platelets from drug-dependent antibodies against platelet glycoproteins. We report the case of a 57-year-old man who developed severe thrombocytopenia and mucocutaneous bleeding following a short course of trimethoprim-sulfamethoxazole (TMP-SMX) for presumed tick-borne disease. The patient experienced bleeding gums, pinpoint rashes, bruising, and extreme fatigue.
View Article and Find Full Text PDFInt J Surg Case Rep
September 2025
Introduction: Duodenal injuries occur in significant number of patients after abdominal trauma. Though most, 75 %, occur after penetrating mechanism of injury the remaining occur after blunt trauma and these are the commonly missed cases unless high index of suspicion is maintained. Here we presented a case of isolated retroperitoneal duodenal perforation after blunt abdominal trauma with a delayed diagnosis and management.
View Article and Find Full Text PDFWorld J Pediatr Congenit Heart Surg
September 2025
Congenital Heart Center, Departments of Surgery and Pediatrics, University of Florida, Gainesville, FL, USA.
This analysis evaluates the longitudinal impact of extracorporeal membrane oxygenation (ECMO) and ventricular assist device (VAD) on the progression of motor delay and cognitive delay in pediatric heart transplant recipients. The United Network for Organ Sharing Registry was queried for pediatric patients (<18 years) who received a heart transplant between 2008 and 2022 and were bridged-to-transplantation with either ECMO or VAD. Patients were further stratified based on the progression of delay status pretransplant to post-transplant.
View Article and Find Full Text PDFNeurocrit Care
September 2025
Department of Paediatrics, Cambridge University, Cambridge, UK.
Background: Low cerebral perfusion pressure (CPP) has previously been identified as a key prognostic marker after pediatric traumatic brain injury (TBI). Cerebrovascular autoregulation supports stabilization of cerebral blood flow within the autoregulation range. Beyond the upper limit of this range, cerebral blood flow increases with increasing CPP, leading to increased risk of intracranial hypertension and blood-brain barrier disruptions.
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