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This study investigated associations of gross motor delay and dyadic emotional availability (EA). Data were drawn from 99 children (Mean age = 8.79 [SD = 3.2] months, 47% Female, and 67% White) with and without motor delay and their caregivers. EA was quantified from five parent-child interactions collected over 12 months. Dyads with children with significant motor delay had significantly less growth in Total EA, Child EA, Sensitivity, Responsiveness, and Involvement compared to dyads with children with typical motor development. Children with mild motor delay differed from children with typical motor development in Involvement. Severity of motor delay was associated with risk to EA in that dyads of children with more significant motor delay had more difficulties with EA over time. Trial Registration: This trial is registered at ClinicalTrials.gov (identifier: NCT02593825).
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http://dx.doi.org/10.1111/infa.70027 | DOI Listing |
Psychol Res
September 2025
Neurorehabilitation Research Center, Kio University, Nara, Japan.
The ability to detect small errors between sensory prediction in the brain and actual sensory feedback is important in rehabilitation after brain injury, where motor function needs to be restored. To date in the recent study, a delayed visual error detection task during upper limb movement was used to measure this ability for healthy participants or patients. However, this ability during walking, which is the most sought-after in brain-injured patients, was unclear.
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August 2025
Department of Speech-Language Pathology and Audiology, Faculty of Humanities, University of Pretoria, Pretoria.
Background: Small infants face more developmental risks than their full-term peers, necessitating early intervention and long-term monitoring.
Objectives: This study examined the longitudinal developmental and hearing outcomes of small infants attending a high-risk clinic in a South African low-income community setting.
Method: A short-term longitudinal within-subject descriptive study design was employed, where 28 participants underwent hearing and developmental screenings and assessments at two follow-up appointments (T1 and T2), at 6- and 12-month corrected age.
Brain Behav
September 2025
Centre For Cognitive and Clinical Neuroscience, College of Health, Medicine and Life Sciences, Brunel University of London, London, UK.
Introduction: There is an ongoing debate about the neural mechanisms and subjective preferences involved in the processing of social rewards compared to non-social reward types.
Methods: Using whole-brain functional magnetic resonance imaging (fMRI), we examined brain activation patterns during the anticipation and consumption phases of monetary and social rewards (using the Monetary and Social Incentive Delay Task-MSIDT, featuring human avatars) and their associations with self-reported social reward preferences measured by the Social Reward Questionnaire (SRQ) in 20 healthy right-handed individuals.
Results: In the anticipation phase, all reward types activated the dorsal striatum, middle cingulo-insular (salience) network, inferior frontal gyrus (IFG), and supplementary motor areas.
Eur J Neurosci
September 2025
Department of Neuroanatomy, Yokohama City University School of Medicine, Yokohama, Japan.
Pelvic visceromotor functions such as micturition are regulated by coordinated autonomic and somatic motor pathways from the central nervous system. The parasympathetic system induces detrusor muscle contraction while the somatic system facilitates relaxation of the external urethral sphincter, ensuring synchronized and efficient bladder emptying during the voiding process. This study explores the relationship between Barrington's nucleus corticotropin-releasing hormone (CRH)-ergic projections and the formation of perineural nets (PNNs) among spinal motoneurons, particularly parasympathetic preganglionic neurons in the intermediolateral nucleus (IML) and Onuf's nucleus during the maturation of the neural circuitry controlling pelvic visceromotor functions.
View Article and Find Full Text PDFMedicine (Baltimore)
September 2025
Diagnosis and Treatment Center for Children, The Affiliated Hospital to Changchun University of Chinese Medicine, Changchun, Jilin Province, China.
Rationale: Phelan-McDermid syndrome, also known as chromosome 22q13.3 deletion syndrome, is a genetic disorder primarily caused by a chromosome 22q13.3 deletion or mutation.
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