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The extent to which latent profiles of psychopathy manifest across the full spectrum of psychopathy (i.e., general population) is unknown. The present study sought to address this gap by subtyping adults based on psychopathic features and exploring whether these profiles differ based on external correlates: motivational tendencies, impulsivity, triarchic psychopathy, anxiety/depression and personality traits. A demographically representative sample of US adults (N = 446; M = 46.10; 51% female; 78.0% White) completed measures of four-factor psychopathy, motivational tendencies, impulsivity, anxiety/depression and personality traits. We identified four latent profiles of psychopathy that were conceptually similar to profiles reported in male offender samples. The impulsive-antisocial profile (i.e., high antisocial behaviour, erratic lifestyle; comparatively lower interpersonal manipulation, callous affect) was characterized by lack of premeditation, sensation seeking, low behavioural inhibition and reduced reward responsiveness when compared to the non-antisocial psychopathic profile (i.e., high interpersonal manipulation, callous affect, erratic lifestyle; comparatively lower scores on antisocial behaviour). Overall, we found evidence in favour of the suitability of self-reported psychopathy to profile individuals in the general population and its ability to distinguish between these subtypes on theoretically relevant external variables.
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http://dx.doi.org/10.1111/bjop.12789 | DOI Listing |
J Behav Med
September 2025
Department of Psychology, University of Wisconsin-La Crosse, La Crosse, WI, USA.
Latent profile analysis (LPA) is in the finite mixture model analysis family and identifies subgroups by participants' responses to continuous variables (i.e., indicators); participants' probable membership in each subgroup is based on the similarity between the subgroup's prototypical responses and the person's unique responses.
View Article and Find Full Text PDFCancer Epidemiol Biomarkers Prev
September 2025
University of Iowa Holden Comprehensive Cancer Center, Iowa City, IA, United States.
Background: Comorbidities may affect incidence and management of cancers. The burden of comorbidities among AIAN cancer patients and survivors is unknown.
Methods: Using SEER-Medicare, we identified AIAN people aged 66+ years diagnosed with female breast, lung, and colorectal cancers (2000-2019), with at least one year of Medicare coverage prior to diagnosis.
J Child Psychol Psychiatry
September 2025
Institute of Psychiatry, Psychology & Neuroscience, King's College London, London, UK.
Background: Prospective studies of autism family history infants primarily report recurrence and predictors of autism at 3 years. Less is known about ADHD family history infants and later childhood outcomes. We characterise profiles of mid-childhood developmental and behavioural outcomes in infants with a family history of autism and/or ADHD to identify potential support needs and patterns of co-occurrence across domains.
View Article and Find Full Text PDFJAACAP Open
September 2025
A.J. Drexel Autism Institute at Drexel University, Philadelphia, Pennsylvania.
Objective: The goal of this study is to characterize health outcomes across 3 domains-overall well-being, behavioral health, and physical health-in a large sample of autistic and non-autistic children and adolescents in the Environmental influences on Child Health Outcomes (ECHO) program.
Method: First, we examined differences in health outcomes between autistic (N = 286) and non-autistic (N = 4,225) children and adolescents in the ECHO Program. Using a subsample of 1,809 participants (116 autistic participants) with complete outcome data, we conducted latent profile analyses (LPAs) to define profiles of health outcomes for autistic children and adolescents and for the combined sample of autistic and non-autistic participants.
JAACAP Open
September 2025
Columbia University, New York, New York.
Objective: The serotonin system has long been implicated in autism spectrum disorder. A previous study reported lower whole blood serotonin (WB5-HT) concentrations in the mothers of children with more severe autism. This study attempted to replicate this finding in an independent cohort.
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