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In recent years, the research on abnormal events detection is a significant work in surveillance video. Many researchers have been attracted by this work for the past two decades. As a result, several abnormal event detection approaches have been developed. Though several approaches have been used in the field still many problems remain to get the abnormal events detection accuracy. Moreover, many feature representations have limited capability to describe the content since several research works applied hand craft features, this type of feature can work in limited problems. To overcome this problem, this paper introduced the novel feature descriptor namely STS-D (Spatial and Temporal Saliency - Descriptor), which includes spatial and temporal information of the objects. This feature descriptor efficiently describes the shape and speed of the object. To find the anomaly score, fuzzy representation is modeled to efficiently differentiate the normal and abnormal events using fuzzy membership degree. The benchmark datasets UMN, UCSD Ped1 and Ped2 and real time roadway surveillance dataset are used to evaluate the performance of the proposed approach. Also, several existing abnormal events detection approaches are used to compare with the proposed method to evaluate the effectiveness of the proposed work.
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http://dx.doi.org/10.1038/s41598-024-81387-x | DOI Listing |
J Neurosci Methods
September 2025
Department of Computer Science and Engineering, IIT (ISM) Dhanbad, Dhanbad, 826004, Jharkhand, India. Electronic address:
Background: Interpretation of motor imagery (MI) in brain-computer interface (BCI) applications is largely driven by the use of electroencephalography (EEG) signals. However, precise classification in stroke patients remains challenging due to variability, non-stationarity, and abnormal EEG patterns.
New Methods: To address these challenges, an integrated architecture is proposed, combining multi-domain feature extraction with evolutionary optimization for enhanced EEG-based MI classification.
Women Birth
September 2025
Department of Women's and Children's Health, Uppsala University, Uppsala, Sweden; Department of Nursing, Mid Sweden University, Sundsvall, Sweden.
Background: Few parents experience stillbirth in Sweden, and their perspectives on the grieving process remain largely unknown.
Objective: To explore parents' perspectives, memories, reflections and insights in the grieving and recovery process six months after stillbirth.
Methods: A mixed-method study involving nine in-depth interviews and responses to eleven quantitative statements.
Ann Surg
September 2025
Department of Surgery, University of Wisconsin-Madison.
Objective: We evaluated the empirical fit of our model of clinical momentum for older adults with life-limiting illness undergoing unplanned surgery.
Background: Older adults often undergo surgery near the end of life, in contrast to generally stated preferences. Systems forces promoting intervention may produce nonbeneficial treatment despite advances in communication.
Rev Cardiovasc Med
August 2025
The Heart Institute, Department of Pediatrics, University of Tennessee Health and Science Center, Memphis, TN 38103, USA.
Left ventricular noncompaction (LVNC), also called noncompaction cardiomyopathy (NCM), is a myocardial disease that affects children and adults. Morphological features of LVNC include a noncompacted spongiform myocardium due to the presence of excessive trabeculations and deep recesses between prominent trabeculae. Incidence and prevalence rates of this disease remain contentious due to varying clinical phenotypes, ranging from an asymptomatic phenotype to fulminant heart failure, cardiac dysrhythmias, and sudden death.
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August 2025
Department of Neurology, Yuhuangding Hospital Affiliated to Qingdao University, Yantai, China.
Essential thrombocythemia (ET) is a myeloproliferative neoplasm (MPN) characterized by abnormal megakaryocyte proliferation and a markedly elevated platelet count, which predisposes patients to thrombotic or hemorrhagic events. Approximately 50%-60% of ET patients harbor a JAK2 V617F mutation. This mutation drives constitutive JAK kinase activation, promoting megakaryocyte proliferation and platelet production, while potentially activating inflammatory pathways and damaging vascular endothelium.
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