Category Ranking
98%
Total Visits
921
Avg Visit Duration
2 minutes
Citations
20
Article Abstract
Neuropathic deformities of the lesser toes refer to a variety of neuromuscular disorders in which a stiff lesser toe deformity is encountered. Claw toes, hammer toes, and mallet toes can be found. Each deformity is better described as which joint of a particular toe is in a flexed or extended position. This information guides the surgeon to which joint should be addressed and what additional soft tissue procedure may be needed. Interphalangeal joint fusions are the procedure of choice, together with extensor or flexor tenotomies. Good results can be expected, with low revision rates.
Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1016/j.fcl.2024.02.001 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Aβ low threshold mechanoreceptors contribute to sensory abnormalities in fibromyalgia.
Brain
September 2025
Wolfson Sensory, Pain and Regeneration Centre, Institute of Psychiatry, Psychology & Neuroscience, King's College London, London, SE1 1UL, UK.
Fibromyalgia syndrome (FM) is characterized by widespread pain and fatigue. People living with FM also experience tactile allodynia, cold-evoked pain, paresthesia and dysesthesia. There is evidence of small fiber neuropathy and hyperexcitability of nociceptors in FM, however the presence of other sensory abnormalities suggests involvement of large diameter sensory fibers.
View Article and Find Full Text PDFThe effect of recombinant botulinum neurotoxin A on neuropathic pain in the spared nerve injury mouse model.
J Neural Transm (Vienna)
August 2025
Core Center for Molecular Morphology, Section for Stereology and Microscopy, Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.
Neuropathic pain following traumatic nerve injury is a disabling chronic pain disorder characterized by sensory abnormalities such as mechanical allodynia. Botulinum neurotoxin type A (BoNT/A) has shown analgesic properties in a range of clinical pain conditions and in animal models. Here, we investigated analgesic efficacy of recombinant BoNT/A1 (rBoNT/A1; IPN10260) in the spared nerve injury (SNI) mouse model of neuropathic pain.
View Article and Find Full Text PDFThe alteration of the sensorimotor network in trigeminal neuralgia after microvascular decompression surgery: a follow-up study using independent component analysis.
Front Physiol
August 2025
Shenzhen Clinical Research Center for Mental Disorders, Shenzhen Kangning Hospital, Shenzhen Mental Health Center, Shenzhen, China.
Introduction: Trigeminal neuralgia (TN) is a chronic neuropathic pain disorder characterized by spontaneous or triggered electric shock-like facial pain. Microvascular decompression (MVD) is the most effective surgical intervention for classical TN that is refractory to medication. Recent advances in neuroimaging have enhanced visualization of the trigeminal nerve's vascular anatomy, deepening insights into TN pathophysiology and paving the way for improved diagnostics and therapies.
View Article and Find Full Text PDFDeclawing in Cat is associated with neuroplastic sensitization and long-term painful afflictions.
Sci Rep
August 2025
Groupe de recherche en pharmacologie animale du Québec (GREPAQ), Université de Montréal, Saint-Hyacinthe, QC, Canada.
Declawing of pet cats is widely believed to cause chronic pain and negatively impact animal welfare, leading to bans in many jurisdictions. However, little is known on how post-declaw pain develops and affects feline well-being. Existing data often fail to account for other sources of chronic pain, such as osteoarthritis (OA), which affects most aging cats.
View Article and Find Full Text PDFMitochondrial Trifunctional Protein Deficiency due to HADHA Variants Masquerading as Charcot-Marie-Tooth Disease.
J Peripher Nerv Syst
September 2025
Department of Neurology, Tallaght University Hospital, Dublin, Ireland.
Background And Aims: Mitochondrial trifunctional protein deficiency (MTPD) is an inherited disorder of fatty acid β-oxidation caused by mutations in HADHA or HADHB genes. It typically presents with cardiomyopathy or hepatic failure in early childhood; however, it may rarely present in adulthood with the neuromyopathic form.
Methods: We describe a patient with MTPD with isolated neuropathy mimicking Charcot-Marie-Tooth disease (CMT) as the first and only presenting symptom.