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Article Abstract
Many patients with antiphospholipid syndrome had decreased ectonucleotidase activity on neutrophils and platelets, which enabled extracellular nucleotides to trigger neutrophil-platelet aggregates. This phenotype was replicated by treating healthy neutrophils and platelets with patient-derived antiphospholipid antibodies or ectonucleotidase inhibitors.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC10972706 | PMC |
| http://dx.doi.org/10.1182/blood.2023022097 | DOI Listing |
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