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http://dx.doi.org/10.1093/jnen/nlab121 | DOI Listing |
Cureus
June 2025
General Physician, GeNeuro - International Research Group in Neuroscience, Vitória, BRA.
Adhesive arachnoiditis (AA) is a rare, chronic inflammatory condition affecting the pia-arachnoid layers, characterized by fibrosis and adhesion formation. It leads to nerve root adhesion and spinal cord compression, often resulting in severe neurological impairment. Commonly associated etiologies include prior spinal surgeries, infections, subarachnoid hemorrhage, epidural anesthesia, myelography, contrast agents, chemical irritation, and, occasionally, idiopathic factors.
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May 2025
Department of Anesthesiology, Texas Tech University Health Sciences Center, Lubbock, TX.
Background: Epidural lysis of adhesions is an effective therapy for treating refractory axial or radicular cervical, thoracic, or lumbar pain. This therapy is an important alternative to surgical interventions. As such, epidural lysis of adhesions is a significant addition to the techniques available to pain management physicians.
View Article and Find Full Text PDFBackground: There is a well-established relationship between liver conditions and cardiovascular diseases. However, uncertainty persists regarding the contribution of liver fibrosis to major stroke types including ischemic stroke, intracerebral hemorrhage, and subarachnoid hemorrhage at the population level.
Methods: In this large prospective cohort study, participants without previous stroke or coronary heart disease at baseline from the UK Biobank were included.
Sci Rep
December 2024
Department of Neurosurgery, Affiliated Hospital of North Sichuan Medical College, Nanchong, 637000, China.
Hydrocephalus commonly occurs after subarachnoid hemorrhage (SAH) and is associated with increased morbidity and disability in patients with SAH. Choroid plexus cerebrospinal fluid (CSF) hypersecretion, obliterative arachnoiditis occluding the arachnoid villi, lymphatic obstruction, subarachnoid fibrosis, and glymphatic system injury are considered the main pathological mechanisms of hydrocephalus after SAH. Although the mechanisms of hydrocephalus after SAH are increasingly being revealed, the clinical prognosis of SAH still has not improved significantly.
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