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Following the publication of this paper [1], it was brought to the authors' attention that one of the contributing authors was left off of the paper. The authors apologize for the unfortunate oversight. In this correction paper, they have included Dr. Paola Tonino in the author list section.
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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7168831 | PMC |
http://dx.doi.org/10.1186/s13395-020-00223-8 | DOI Listing |
J Refract Surg
September 2025
JENVIS Research, Jena, Germany.
Purpose: To analyze the difference in objective and subjective photic phenomena following virtual implantation of three different presbyopia-correcting diffractive intraocular lens (IOL) designs.
Methods: The study was conducted at JENVIS Research Germany. A prospective cross-over and double-masked trial design was used.
JDS Commun
September 2025
Faculté de médecine vétérinaire, Université de Montréal, Saint-Hyacinthe, Canada, J2S 2M2.
The objective of this ambidirectional observational cohort study was to explore how nonesterified fatty acids (NEFA) 22 to 35 d before calving were related to NEFA 1 to 14 d before calving and to determine a threshold that could be used to identify cows at risk of poor postpartum health. We enrolled 855 dairy cows from 46 herds, 362 prospectively and 493 retrospectively. The NEFA concentrations were measured during the far-off period (foNEFA; 3 to 5 wk before calving) and in the close-up period (cuNEFA; up to 2 wk before calving), and postpartum infectious and metabolic disorders, reproduction success, and culling were recorded.
View Article and Find Full Text PDFMol Med
September 2025
Department of Pharmacology, College of Medicine, University of Tennessee Health Science Center, 71 S. Manassas St. Room 225N, Memphis, TN, 38103, USA.
Sci Rep
September 2025
Shanghai Key Laboratory of Molecular Imaging, Shanghai University of Medicine and Health Science, 279Th Zhouzhu Road, Shanghai, 201318, China.
Am J Physiol Cell Physiol
September 2025
Department of Cell Biology, University of Pittsburgh, Pittsburgh, PA.
We previously demonstrated the CFTR correctors VX-445 (elexacaftor) and S-VX-121 (vanzacaftor) potentiate heterologously-expressed BK channels, as well as in primary human bronchial epithelial cells (HBEs). This potentiation of BK resulted in altered vasoreactivity and neuronal excitability. We postulated novel compounds could be identified that would potentiate BK while not affecting CFTR.
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