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Causal queries about singular cases are ubiquitous, yet the question of how we assess whether a particular outcome was actually caused by a specific potential cause turns out to be difficult to answer. Relying on the causal power framework (Cheng, ), Cheng and Novick () proposed a model of causal attribution intended to help answer this question. We challenge this model, both conceptually and empirically. We argue that the central problem of this model is that it treats causal powers that are probabilistically sufficient to generate the effect on a particular occasion as actual causes of the effect, and thus neglects that sufficient causal powers can be preempted in their efficacy. Also, the model does not take into account that reasoners incorporate uncertainty about the underlying general causal structure and strength of causes when making causal inferences. We propose a new measure of causal attribution and embed it into the structure induction model of singular causation (SISC; Stephan & Waldmann, ). Two experiments support the model.
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http://dx.doi.org/10.1111/tops.12309 | DOI Listing |
Cancer Med
July 2025
Department of General Surgery (Thyroid Surgery), The Affiliated Hospital of Southwest Medical University, Luzhou, China.
Aim: To investigate the mechanistic aspects of inflammation and oxidative stress and their association with thyroid cancer risk.
Methods: We have systematically searched the PubMed and Web of Science databases to perform a comprehensive analysis of the pathogenic mechanisms that link obesity, inflammation, and oxidative stress to thyroid cancer.
Results: Chronic inflammation, a well-known risk factor for cancer progression, is a hallmark characteristic of obesity.
J Gastrointest Surg
September 2025
Department of Surgery, The Ohio State University Wexner Medical Center and James Comprehensive Cancer Center, Columbus, OH, United States. Electronic address:
Background: Among patients with hepatocellular carcinoma (HCC), late recurrence - defined as recurrence occurring ≥2 years after treatment - has often been treated as a singular, uniform event, despite being inherently heterogeneous and driven by diverse biologic mechanisms. This study aimed to identify prognostic factors associated with recurrence among long-term survivors of HCC after treatment, with particular emphasis on the role of underlying liver fibrosis and intrinsic tumor aggressiveness.
Methods: Patients who underwent hepatectomy for HCC between 2000 and 2021 were identified from an international database.
PLoS One
June 2025
Centre for Advancing Health Outcomes, Providence Health Care Research Institute, St. Paul's Hospital, Vancouver, Canada.
Background: Synergistic effects of diverse social supports (informational, tangible, emotional and belonging) on cardiovascular disease risk factors (CVRF), by gender, is unknown.
Aim: To quantify gender differences in the singular and combined associations of four different forms of social support with cardiovascular disease risk factors (CVRF) in aging adults.
Methods: Cross-sectional study of 28,779 adults (45-85 years) in the Canadian Longitudinal Study on Aging Comprehensive cohort (2011-15); independent variables were self-reported measures of informational, tangible, emotional and belonging support; dependent variables were clinically measured BMI, waist circumference and blood pressure.
J Exp Biol
July 2025
Division of Fluid Dynamics, Department of Mechanics and Maritime Sciences, Chalmers University of Technology, 412 96 Gothenburg, Sweden.
In insect locomotion, the transmission of energy from muscles to motion is a process within which there are many sources of dissipation. One significant but understudied source is the structural damping within the insect exoskeleton itself: the thorax and limbs. Experimental evidence suggests that exoskeletal damping shows frequency (or rate) independence, but investigation into its nature and implications has been hampered by a lack methods for simulating the time-domain behaviour of this damping.
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May 2025
Agalma Foundation, Geneva, Switzerland.
This article introduces a model of dialogue between psychoanalysis and neuroscience that is based on an account of the economic dimension of trauma. From the outset Freudian theory took into account the singularity of each subject's response to traumatic events, setting aside any linear paradigm in the causality of symptoms. In 1980, the introduction of the nosographic category of PTSD (post-traumatic stress disorder) within the DSM (Diagnostic and Statistical Manual) contributed to an increased social recognition for sufferers.
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