Publications by authors named "P Padmapriya"

Temporary disturbances in brain function are caused by epilepsy, a chronic disorder resulting from sudden abnormal firing of brain neurons. This research introduces an innovative real-time methodology representing detecting epileptic spasms from electroencephalogram (EEG) data. It employs a support vector machine (SVM) alongside embedded zero tree wavelet (EZW) transform.

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Purpose: We aim to look at the clinical profile in paediatric ocular sarcoidosis in a tertiary eye centre from South India.

Methods: Retrospective study between 2012 and 2024.

Results: A total of 29 cases of paediatric ocular sarcoidosis including 19 cases of late onset sarcoid and 10 cases of early onset sarcoid/Blau syndrome were retrieved from our medical database.

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Article Synopsis
  • The study evaluated the effectiveness of biologic treatments for noninfectious uveitis and other eye inflammatory diseases in a South Indian population over several years.
  • A total of 76 patients, predominantly adults with anterior nongranulomatous uveitis, were analyzed, revealing adalimumab as the most frequently used biologic.
  • While biologics led to a significant reduction in flare-ups, their success rates were lower for certain conditions, particularly in younger patients with specific types of uveitis.
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Purpose: To report a rare presentation of a proven case of infection presenting as multifocal choroiditis with recurrent choroidal neovascular membrane (CNVM) in one eye, initially misdiagnosed as punctate inner choroidopathy and later developed serpiginous-like choroiditis in the other eye.

Methods: Retrospective case report with a review of existing literature.

Results: A 30-year-old women presented with metamorphopsia (OD) and best-corrected visual acuity (BCVA) of 6/24 (OD) and was diagnosed to have punctate inner choroidopathy with CNVM (OD).

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The distinct disease progression patterns of severe acute respiratory syndrome coronavirus clade 2 (SARS-CoV-2) indicate diverse host immune responses. SARS-CoV-2 severely impairs type I interferon (IFN) cell signaling, resulting in uncontrolled late-phase lung damage in patients. For better pharmacological properties, cytokine modifications may sometimes result in a loss of biological activity against the virus.

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