Long-term Exposure to Oxidant Gases and Mortality: Effect Modification by PM 2.5 Transition Metals and Oxidative Potential.

Background: Populations are simultaneously exposed to outdoor concentrations of oxidant gases (i.e., O 3 and NO 2 ) and fine particulate air pollution (PM 2.

Association of Sulfur, Transition Metals, and the Oxidative Potential of Outdoor with Acute Cardiovascular Events: A Case-Crossover Study of Canadian Adults.

Background: We do not currently understand how spatiotemporal variations in the composition of fine particulate air pollution [fine particulate matter with aerodynamic diameter ()] affects population health risks. However, recent evidence suggests that joint concentrations of transition metals and sulfate may influence the oxidative potential (OP) of and associated health impacts.

Objectives: The purpose of the study was to evaluate how of transition metals/OP and sulfur content in outdoor influence associations with acute cardiovascular events.

Living near major roads and the incidence of dementia, Parkinson's disease, and multiple sclerosis: a population-based cohort study.

Background: Emerging evidence suggests that living near major roads might adversely affect cognition. However, little is known about its relationship with the incidence of dementia, Parkinson's disease, and multiple sclerosis. We aimed to investigate the association between residential proximity to major roadways and the incidence of these three neurological diseases in Ontario, Canada.

A New Method to Jointly Estimate the Mortality Risk of Long-Term Exposure to Fine Particulate Matter and its Components.

Most studies on the association between exposure to fine particulate matter (PM2.5) and mortality have considered only total concentration of PM2.5 or individual components of PM2.

The impact of a landfill fire on ambient air quality in the north: A case study in Iqaluit, Canada.

A large landfill fire occurred in Iqaluit, Canada in spring/summer 2014. Air quality data were collected to characterize emissions as well as potential threats to public health. Criteria pollutants were monitored (PM2.

Tracking national and regional spatial-temporal mortality risk associated with NO2 concentrations in Canada: a Bayesian hierarchical two-level model.

The association between daily variations in urban air quality and mortality has been well documented using time series statistical methods. This approach assumes a constant association over time. We develop a space-time dynamic model that relaxes this assumption, thus more directly examining the hypothesis that improvements in air quality translate into improvements in public health.

A temporal, multicity model to estimate the effects of short-term exposure to ambient air pollution on health.

Background: Countries worldwide are expending significant resources to improve air quality partly to improve the health of their citizens. Are these societal expenditures improving public health?

Objectives: We consider these issues by tracking the risk of death associated with outdoor air pollution over both space and time in Canadian cities.

Materials And Methods: We propose two multi-year estimators that use current plus several previous years of data to estimate current year risk.

Measuring progress in the management of ambient air quality: the case for population health.

Although progress has been made in the last few decades at reducing ambient concentrations of air pollutants, scientific evidence suggests that there remains a risk to human health from exposure to these pollutants at current levels in Canada. Much of the motivation for air pollution reduction efforts is to protect population health. This article presents a method of monitoring changes in air pollution-related health outcomes over time in conjunction with temporal changes in ambient pollution concentrations.

Identifying and managing adverse environmental health effects: 6. Carbon monoxide poisoning.

Carbon monoxide poisoning is an enigmatic illness. The symptoms are often non-specific or masked by an exacerbation of an underlying illness, such as congestive heart failure, that has been triggered by carbon monoxide inhalation. The effects can range from mild, annoying symptoms relieved by removal of the source to severe morbidity with profound central nervous system dysfunction, acute complications and delayed sequelae.