Observation of Three Resonant Structures in the Cross Section of e^{+}e^{-}→π^{+}π^{-}h_{c}.

Using e^{+}e^{-} collision data collected with the BESIII detector operating at the Beijing electron positron collider, the cross section of e^{+}e^{-}→π^{+}π^{-}h_{c} is measured at 59 points with center-of-mass energy sqrt[s] ranging from 4.009 to 4.950 GeV with a total integrated luminosity of 22.

Mitochondrial transfer from adipose-derived regenerative cells contributes therapeutic angiogenesis in a murine hindlimb ischemia model.

Objective: Adipose-derived regenerative cells (ADRCs) are promising cell sources for damaged tissue regeneration. The efficacy of therapeutic angiogenesis with ADRC implantation in patients with critical limb ischemia has been demonstrated in clinical studies. There are several possible mechanisms in this process such as cytokines and microRNA.

Associations of dietary mineral intakes with the risk of six common mental disorders: A prospective cohort study.

Background: Evidence on the relationship between dietary minerals and mental disorders remains limited and inconsistent. This study assessed the associations between twelve essential minerals and six major mental disorders.

Methods: We included 199,877 participants from the UK biobank without implausible energy intake, missing covariates, or baseline mental disorders.

On the species status of and (Blattodea, Blattellidae), and other morphologically similar species.

(Linnaeus, 1767), a member of the Blattellidae family within the order Blattodea, is a significant global sanitary pest. Several species within the genus Caudell, 1903 and its closely related genera ( Bey-Bienko, 1950; Hebard, 1916; and Hebard, 1929) exhibit external morphological traits similar to those of . By integrating morphological identification and molecular analyses, one new species was identified: Cai, Yao & Che, Additionally, Mizukubo, 1981 was downgraded to a subspecies of .

Advance in molecular mechanisms underlying diabetes related to viral hepatitis infection.

Diabetes and viral hepatitis, particularly hepatitis B (HBV) and hepatitis C (HCV), are significant global health burdens with complex interconnections. This review discusses the molecular mechanisms linking viral hepatitis to diabetes, focusing on inflammatory pathways, oxidative stress, and epigenetic modifications. Key findings highlight the role of STAT3 in promoting insulin resistance and β-cell apoptosis, the impact of ER stress and NOX-mediated oxidative stress on metabolic dysfunction, and the influence of epigenetic changes such as DNA methylation and histone acetylation on glucose homeostasis.